O negative is the universal donor I believe because it has no antigens. My dad is O neg and he gives blood as often as possible because he was once told it is often given to preemies. No idea if that is actually true or if he's just a sucker.
Post by Norticprincess on May 6, 2013 11:24:59 GMT -5
I'm not sure if I'm A+ or O + at the moment. Prior to April 12th I was 100% A+. My sister is O+ and is my BMT donor. Things are in the process of changing. My last red blood transfusion was more than a week ago and they stuck with O products. I haven't seen the results of my typing recently. Docs said it could take a few months for everything to even out.
She is much more adventurous with food, eats more dairy and spicy things than I ever do.
"Why would you ruin perfectly good peanuts by adding candy corn? That's like saying hey, I have these awesome nachos, guess I better add some dryer lint." - Nonny
"Why would you ruin perfectly good peanuts by adding candy corn? That's like saying hey, I have these awesome nachos, guess I better add some dryer lint." - Nonny
"Why would you ruin perfectly good peanuts by adding candy corn? That's like saying hey, I have these awesome nachos, guess I better add some dryer lint." - Nonny
System Gene Polymorphism SNP⁎ Amino acid change† ABO ABO A/B 526C > G, 703G > A, 796C > A, 803G > C R176G, G235S, L266M, G268A
A and B transferases are encoded by a single gene on chromosome 9. The ABO gene spans about 18–20 kilobases (kb) organised into seven exons. Exons 6 and 7, the two largest, encode 77% of the full coding region and probably the entire domain responsible for catalytic activity.
The products of the A and B alleles differ by four amino acid substitutions: Arg176Gly, Gly235Ser, Leu266Met, and Gly268Ala [7] (Fig. 2). Amino acid residues at positions 266 and 268 are most important in determining whether the gene product is an A-transferase (N-acetylgalactosaminyltransferase) or B-transferase (galactosyltransferase). The sequence of the most common O allele (O1, also known as O01) is identical to that of the A sequence, apart from the deletion of a single nucleotide in exon 6. This nucleotide deletion is responsible for a reading-frame shift generating a translation stop signal at codon 117 [7] (Fig. 2). This allele encodes a truncated protein lacking the catalytic site. Another common O allele, O1v (O02), also has the single nucleotide deletion, but differs from O1 by nine nucleotide changes within the coding sequence. Functionally, O1 and O1v are identical. The O2 (O03) allele, which represents about 3% of O genes in a Caucasian population, does not have the single nucleotide deletion but is inactivated by a mutation that encodes a substitution of Gly268 by arginine. The O2 gene product has Leu266 (typical of an A-transferase), and therefore cannot transfer galactose, but the Arg268 blocks access of N-acetylgalactosamine, so neither A nor B antigen is synthesised [8] (Fig. 2). At least 60 other rare O alleles, with inactive products, have been identified. For reviews on ABO see references [9] and [10].
I'm B- and feel oh so all alone in here. My husband is O- and so is my daughter. We actually just discussed this the last time I visited family. Neither of my brothers knew what blood types they were. Then I realized I knew it from being in the military (and have also had tons of blood drawn because of it).
Hmmmm. Well, here's what I think. I think that there are 20,000 genes in the human genome that code for proteins, and the ABO blood type is just one or two of those. So I can say that absolutely without a single doubt I feel better without dairy in my life. And I'm A- blood type. Can I say that the reason for that is the ABO gene vs any one of the other 19,998? No, probably not. We are just too damn complicated for something that simple. It might be a factor, but there are 20,000 other potential factors to take into account. I don't think it's a bad place to start, especially if you are searching for an answer, but I think there are many many more factors in play than just the blood type gene. And some of them aren't even gene related! You have to take the gut microflora into consideration, and that's not even accounted for by your genes. And then there is something called epigenetics, where your genes are modified, but not due to changes in the sequence itself.
Basically, the fact that there are living breathing digesting procreating human beings walking around makes me look like signature cat
I think I'm supposed to eat a lot of fish and limit dairy.
But if I pip my fridge ( which I won't confession: bc it looks like sleeping with the enemy organized) there is a ton of dairy. DAIRY FOR EVERYONE!! WHEEEE
A+ I have quick temper with tendency towards rage. Carbs make me fat. I love carbs and try not to eat them. Which might be the cause of my rage.
Is the fact that you're not eating right for your blood type the cause of your rage?
Ohhh! Half-baked facts and speculations served up nice and hot!
Anyway, you can rage out with me anytime.
For Science: My Mom and sister are on the blood type diet plan. Mom lost 15 lbs and sister lost 5 lbs. Sister only had that to lose. Mom has stalled out and is probably 30 lbs to go.
Post by AlpineSlide on May 6, 2013 11:51:51 GMT -5
I'm O- and can eat anything with no digestive issues.
For those wondering about your blood type - go give blood (if you feel like it) and the Red Cross will tell you your blood type. I think they send a letter to you a week or two later, maybe they can also tell you the same day that you donate but you definitely get something in the mail.